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CJC-1295 No DAC

GH-axis research

CJC-1295 No DAC

Gevriesdroogd endocriene signaleringsonderzoekpeptide voor systeembeoordeling

CJC-1295 No DAC is a short-acting GHRH analog studied as a pituitary GH-axis signaling reference.

Prijsklasse: 34.99 $ tot 259.99 $
98.7%-99.4% Gevriesdroogd poeder 5 mg
Available documentation

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Alleen voor laboratoriumonderzoek. Niet voor menselijk of diergeneeskundig gebruik. Niet bedoeld voor het diagnosticeren, behandelen, genezen, verzachten of voorkomen van ziekten.

Opslag

Afgesloten, koud, droog en beschermd tegen hitteschommelingen bewaren. Bevestig productspecifieke opslag bij aankomst.

Verzendinformatie

Verpakt voor stabiliteit tijdens het transport, met bestelondersteuning beschikbaar voor routes bij warm weer.

Batch reference

Batch-linked certificate support is available through the documentation desk.

Onderzoekscontext

CJC-1295 No DAC onderzoeksprofiel

CJC-1295 No DAC is used as a shorter-acting GHRH analog reference in pituitary GH-axis research. Studies focus on GHRH-receptor activation, stimulated GH release, pulse timing, downstream IGF-1 markers, and comparisons with longer-acting analogs. The “No DAC” designation separates this material from CJC-1295 versions designed with a drug-affinity complex for albumin binding and prolonged exposure. It is generally selected when the experimental design values shorter signaling windows or direct comparison with sermorelin and secretagogue compounds. Nomenclature for modified GRF and CJC-related peptides can vary, so sequence identity, amount, and batch documents are important parts of product selection.

Molecular class

Modified 29-residue GHRH analog

Sequence / composition

A substituted growth-hormone-releasing-hormone 1-29 analog without a Drug Affinity Complex attachment.

Research design note

The absence of DAC is central to the shorter-exposure pharmacology usually compared in GH-axis studies.

Voorgesteld mechanisme

A short-acting GHRH-receptor agonist concept, often associated in commerce with modified GRF(1-29). GHRH receptor activation increases cyclic AMP in pituitary somatotrophs and promotes pulsatile growth-hormone release.

In studies gerapporteerde effecten

  • GHRH analog studies report acute increases in GH release and downstream IGF-1-related signaling, depending on analog and exposure.
  • Shorter exposure is expected to produce a different release profile from albumin-binding CJC-1295 with DAC.
  • The label "CJC-1295 No DAC" is used inconsistently and may not describe the same sequence studied under the original CJC-1295 name.

Veelgebruikte onderzoekseindpunten

Sequence confirmation, GHRH receptor activity, cyclic AMP, GH pulse amplitude and duration, IGF-1, pituitary responsiveness, degradation, and comparison with DAC-modified material.

Bewijs en beperkingen

GHRH biology is established, but direct evidence is weakened by naming and identity ambiguity. Sequence and batch documentation are especially important.

External reading

Literature for context—not product proof.

Third-party sources describe their own research materials and methods. They do not validate a specific Azure catalog batch.

Support and documentation

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